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Epimutations in both the TESK2 and MMACHC promoters in the Epi-cblC inherited disorder of intracellular metabolism of vitamin B 12 .

Abderrahim OussalahYoussef SibliniSébastien HergalantCéline ChéryPierre RouyerCatia CavicchiRenzo GuerriniPierre-Emmanuel MorangeDavid TrégouëtMihaela PupavacDavid WatkinsTomi PastinenWendy K ChungCan FiciciogluFrançois FeilletD Sean FroeseMatthias R BaumgartnerJean-François BenoistJacek MajewskiAmelia MorroneDavid S RosenblattJean-Louis Guéant
Published in: Clinical epigenetics (2022)
The antisense readthrough transcription of the mutated PRDX1 produces an epigenetic silencing of MMACHC and TESK2. We propose using the term 'epi-digenism' to define this epigenetic disorder that affects two genes. Epi-cblC is an entity that differs from cblC. Indeed, the PRDX1 and TESK2 altered expressions are observed in epi-cblC but not in cblC, suggesting further evaluating the potential consequences on cancer risk and spermatogenesis.
Keyphrases
  • dna methylation
  • gene expression
  • preterm infants
  • risk assessment
  • human health
  • genome wide analysis