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Correction: Targeting the overexpressed mitochondrial protein VDAC1 in a mouse model of Alzheimer's disease protects against mitochondrial dysfunction and mitigates brain pathology.

Ankit VermaAnna Shteinfer-KuzmineNikita KamenetskySrinivas PittalaAvijit PaulEdna Nahon CrystalAlberto OuroVered Chalifa-CaspiSwaroop Kumar PandeyAlon MonsonegoNoga VardiShira KnafoVarda Shoshan-Barmaz
Published in: Translational neurodegeneration (2023)
Keyphrases
  • mouse model
  • oxidative stress
  • resting state
  • white matter
  • cognitive decline
  • cancer therapy
  • protein protein
  • functional connectivity
  • binding protein
  • cerebral ischemia
  • multiple sclerosis
  • drug delivery