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Loss of tumor protein 53 protects against alcohol-induced facial malformations in mice and zebrafish.

Eric W FishScott K TuckerRachel L PetersonJohann K EberhartScott E Parnell
Published in: Alcoholism, clinical and experimental research (2021)
These results identify an evolutionarily conserved role of Tp53 as a pathogenic mechanism for alcohol-induced teratogenesis.
Keyphrases
  • high glucose
  • diabetic rats
  • drug induced
  • transcription factor
  • alcohol consumption
  • type diabetes
  • metabolic syndrome
  • skeletal muscle
  • insulin resistance
  • binding protein
  • stress induced