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Activation of CD137 Signaling Enhances Vascular Calcification through c-Jun N-Terminal Kinase-Dependent Disruption of Autophagic Flux.

Rui ChenYao XuWei ZhongBo LiPing YangZhong Qun WangChen ShaoCui Ping WangJin Chuan Yan
Published in: Mediators of inflammation (2018)
The present study identified a new pathway in which CD137 promotes VSMC calcification through the activation of JNK signaling, subsequently leading to the disruption of autophagic flux, which is responsible for CD137-induced acceleration of vascular calcification.
Keyphrases
  • cell death
  • chronic kidney disease
  • nk cells
  • signaling pathway
  • diabetic rats
  • tyrosine kinase
  • endoplasmic reticulum stress