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Snail acetylation by autophagy-derived acetyl-coenzyme A promotes invasion and metastasis of KRAS-LKB1 co-mutated lung cancer cells.

Jang Hee HanYong Keon KimHakhyun KimJooyoung LeeMyung Joon OhSang Bum KimMinjee KimKook Hwan KimHyun Ju YoonMyung-Shik LeeJohn D MinnaMichael A WhiteHyun Seok Kim
Published in: Cancer communications (London, England) (2022)
This study demonstrates that autophagy-derived acetyl-CoA promotes Snail acetylation and thereby facilitates invasion and metastasis of KRAS-LKB1 co-mutated lung cancer cells and that inhibition of the autophagy/acetyl-CoA/acetyl-Snail axis using CAMKK2 or ACLY inhibitors could be a potential therapeutic strategy to suppress metastasis of KL lung cancer.
Keyphrases
  • epithelial mesenchymal transition
  • cell death
  • endoplasmic reticulum stress
  • signaling pathway
  • wild type
  • oxidative stress
  • cell migration
  • histone deacetylase