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Genome-wide p63-Target Gene Analyses Reveal TAp63/NRF2-Dependent Oxidative Stress Responses.

Marco NapoliAvani A DeshpandeDeepavali ChakravartiKimal I RajapakshePreethi H GunaratneCristian CoarfaElsa R Flores
Published in: Cancer research communications (2024)
The p63 isoforms, TAp63 and ΔNp63, control epithelial morphogenesis and tumorigenesis through the interaction with distinct transcription factors and the subsequent regulation of unique transcriptional programs.
Keyphrases
  • genome wide
  • transcription factor
  • dna methylation
  • copy number
  • oxidative stress
  • gene expression
  • genome wide identification
  • public health
  • dna binding