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Ecto-CD38-NADase inhibition modulates cardiac metabolism and protects mice against Doxorubicin-induced cardiotoxicity.

Thais R PeclatGuillermo AgorrodyLaura ColmanSonu KashyapJulianna D ZeidlerClaudia Christiano Silva ChiniGina M WarnerKatie L ThompsonPranjali DalviFelipe Cesar BeckedorffSanam EbtehajJoerg HerrmannWim van SchootenEduardo Nunes Chini
Published in: Cardiovascular research (2024)
Specific and non-cytotoxic ecto-NADase anti-CD38 antibodies may be a potential therapy for anthracyclines-induced cardiac dysfunction.
Keyphrases
  • high glucose
  • diabetic rats
  • left ventricular
  • oxidative stress
  • drug induced
  • drug delivery
  • heart failure
  • metabolic syndrome
  • high fat diet induced
  • atrial fibrillation
  • wild type