Elevated liver glycogenolysis mediates higher blood glucose during acute exercise in Barth syndrome.
George G SchweitzerGrace L DitzenbergerCurtis C HugheyBrian N FinckMichael R MartinoChristina A PacakBarry J ByrneWilliam Todd CadePublished in: PloS one (2023)
Our data in the Tafazzin knockdown mouse suggest that elevated anaerobic metabolism during rest and exercise previously reported in humans with BTHS are supported by the finding of higher hepatic glycogenolysis.
Keyphrases
- blood glucose
- high intensity
- physical activity
- liver failure
- glycemic control
- microbial community
- resistance training
- wastewater treatment
- blood pressure
- respiratory failure
- electronic health record
- case report
- type diabetes
- aortic dissection
- metabolic syndrome
- risk assessment
- intensive care unit
- sewage sludge
- hepatitis b virus
- weight loss
- insulin resistance
- extracorporeal membrane oxygenation
- artificial intelligence
- acute respiratory distress syndrome