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EIF3C Promotes Lung Cancer Tumorigenesis by Regulating the APP/HSPA1A/LMNB1 Axis.

Xiaoli DingLanlan HouHuijuan ZhangZhiping ChenZhanyu LiuJunjie GongZhi-Xian TangRong Hu
Published in: Disease markers (2022)
EIF3C overexpression may facilitate the proliferation and hamper the apoptosis of lung cancer cells by regulating the APP/HSPA1A/LMNB1 axis.
Keyphrases
  • heat shock protein
  • oxidative stress
  • endoplasmic reticulum stress
  • signaling pathway
  • cell proliferation
  • cell cycle arrest
  • transcription factor
  • pi k akt