DMT1 knockout abolishes ferroptosis induced mitochondrial dysfunction in C. elegans amyloid β proteotoxicity.
Wilson PengKaitlin B ChungB Paige LawrenceM Kerry O'BanionRobert T DirksenAndrew P WojtovichJohn O OnukwuforPublished in: bioRxiv : the preprint server for biology (2024)
Energetic imbalance is an early event in iron-induced loss of neuronal functionNeuronal Aβ increases susceptibility to ferroptosis mediated oxidative damageDivalent metal transporter 1 knockout protects against iron-induced oxidative damage and ferroptosis.