Subtype-Independent ANP32E Reduction During Breast Cancer Progression in Accordance with Chromatin Relaxation.
Garrett L RuffKristin E MurphyZachary R SmithPaula M VertinoPatrick J MurphyPublished in: BMC cancer (2021)
Our results indicate that ANP32E may function through chromatin state regulation to control breast cancer differentiation and tumor plasticity. This study sets a precedent for future computational studies of chromatin changes in carcinogenesis.