Login / Signup

Subtype-Independent ANP32E Reduction During Breast Cancer Progression in Accordance with Chromatin Relaxation.

Garrett L RuffKristin E MurphyZachary R SmithPaula M VertinoPatrick J Murphy
Published in: BMC cancer (2021)
Our results indicate that ANP32E may function through chromatin state regulation to control breast cancer differentiation and tumor plasticity. This study sets a precedent for future computational studies of chromatin changes in carcinogenesis.
Keyphrases
  • dna damage
  • gene expression
  • transcription factor
  • genome wide
  • dna methylation
  • current status
  • single molecule
  • breast cancer risk
  • childhood cancer