Differential Expression of Inflammarafts in Macrophage Foam Cells and in Nonfoamy Macrophages in Atherosclerotic Lesions.

Juliana M Navia-PelaezColin Agatisa-BoyleSoo-Ho Choi Yi Sak KimShenglin LiElena AlekseevaKimberly WeldyYury I Miller

Published in: Arteriosclerosis, thrombosis, and vascular biology (2022)

Our data support the hypothesis that persistent inflammarafts in nonfoamy macrophages in atherosclerotic lesions serve as effectors of macrophage reprogramming into a hyperinflammatory phenotype.

Keyphrases

induced apoptosis
adipose tissue
cell cycle arrest
electronic health record
big data
oxidative stress
cell death
machine learning
cell proliferation
artificial intelligence
data analysis