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Biological concepts in human sodium channel epilepsies and their relevance in clinical practice.

Andreas BrunklausJuanjiangmeng DuFelix StecklerIsmael I GhantyKatrine Marie JohannesenChristina Dühring FengerStephanie SchorgeDavid Baez-NietoHao-Ran WangAndrew AllenJen Q PanHolger LercheHenrike HeyneJoseph D SymondsSameer M ZuberiStephan SandersBeth R SheidleyDana CraiuHeather E OlsonSarah WeckhuysenPeter DeJongeIngo HelbigHilde Van EschTiffany BusaMatthieu MilhBertrand IsidorChristel DepienneAnnapurna H PoduriArthur J CampbellJordane DimidschsteinRikke Steensjerre MollerDennis Lal
Published in: Epilepsia (2020)
Variant function and location within one channel can serve as a surrogate for variant effects across related sodium channels. Taking a broader view on precision treatment suggests that in those patients with a suspected underlying genetic epilepsy presenting with neonatal or early onset seizures (<3 months), SCBs should be considered.
Keyphrases
  • early onset
  • clinical practice
  • late onset
  • endothelial cells
  • pulmonary embolism
  • induced pluripotent stem cells
  • copy number
  • gene expression
  • temporal lobe epilepsy
  • replacement therapy