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MTOR suppresses autophagy-mediated production of IL25 in allergic airway inflammation.

Wen LiYinfang WuYun ZhaoZhou-Yang LiHaixia ChenLingling DongHuiwen LiuMin ZhangYanping WuJiesen ZhouJuan XiongYue HuWen HuaBin ZhangMinzhi QiuQing-Ling ZhangChunhua WeiMingchun WenJing HanXiaobo ZhouWeiliang QiuFugui YanHuaqiong HuangSongmin YingAugustine M K ChoiHuahao ShenZhihua Chen
Published in: Thorax (2020)
Collectively, these results demonstrate that allergen-initiated inflammation suppresses MTOR and induces autophagy in airway epithelial cells, which results in the production of certain proallergic cytokines such as IL25, further promoting the type 2 response and eventually perpetuating airway inflammation in asthma.
Keyphrases
  • signaling pathway
  • oxidative stress
  • allergic rhinitis
  • cell death
  • endoplasmic reticulum stress
  • cell proliferation
  • chronic obstructive pulmonary disease
  • lung function