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γ-Enolase enhances Trk endosomal trafficking and promotes neurite outgrowth in differentiated SH-SY5Y cells.

Anja PišlarJanko Kos
Published in: Cell communication and signaling : CCS (2021)
These data therefore establish the Trk receptor as a binding partner of γ-enolase, whereby Trk endosomal trafficking is promoted by γ-Eno peptide to mediate its neurotrophic signalling. Video abstract.
Keyphrases
  • induced apoptosis
  • cell cycle arrest
  • electronic health record
  • binding protein
  • endoplasmic reticulum stress
  • oxidative stress
  • cell death
  • dna binding
  • hepatitis c virus
  • data analysis