γ-Enolase enhances Trk endosomal trafficking and promotes neurite outgrowth in differentiated SH-SY5Y cells.

Published in: Cell communication and signaling : CCS (2021)

These data therefore establish the Trk receptor as a binding partner of γ-enolase, whereby Trk endosomal trafficking is promoted by γ-Eno peptide to mediate its neurotrophic signalling. Video abstract.

Keyphrases

induced apoptosis
cell cycle arrest
electronic health record

binding protein
endoplasmic reticulum stress
oxidative stress

cell death
dna binding

hepatitis c virus
data analysis