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Ginsenoside Rh4 prevents endothelial dysfunction as a novel AMPK activator.

Jiawei ZhangWeiqiang LvXuyun LiuZhenyu SunMengqi ZengJiahao KangQi ZhangFuying LiuShaozhou MaXiaolong LiKe CaoJiankang Liu
Published in: British journal of pharmacology (2024)
Rh4 may serve as a novel AMPK activator to protect against hyperlipidaemia-induced endothelial dysfunction, providing new insights into the prevention and treatment of endothelial injury-associated CVD.
Keyphrases
  • nuclear factor
  • skeletal muscle
  • high glucose
  • endothelial cells
  • diabetic rats
  • drug induced
  • mouse model
  • oxidative stress
  • immune response
  • smoking cessation