Hydrogen Sulfide Reduces Ischemia and Reperfusion Injury in Neuronal Cells in a Dose- and Time-Dependent Manner.
Stefanie ScheidMax GoellerWolfgang BaarJakob WollbornHartmut BuerkleGünther SchlunckWolf LagrèzeUlrich GoebelFelix UlbrichPublished in: International journal of molecular sciences (2021)
After neuronal IRI, H2S mediates neuroprotection in a time- and dose-dependent manner. The H2S treatment modulated transcription factor NF-κB activation and reduced retinal inflammation.
Keyphrases
- cerebral ischemia
- transcription factor
- induced apoptosis
- oxidative stress
- subarachnoid hemorrhage
- signaling pathway
- brain injury
- blood brain barrier
- optical coherence tomography
- cell cycle arrest
- diabetic retinopathy
- acute myocardial infarction
- lps induced
- endoplasmic reticulum stress
- combination therapy
- nuclear factor
- acute coronary syndrome
- acute ischemic stroke
- cell proliferation
- atrial fibrillation
- replacement therapy
- smoking cessation