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Dual action of high estradiol doses on MNU-induced prostate neoplasms in a rodent model with high serum testosterone: Protective effect and emergence of unstable epithelial microenvironment.

Bianca F GonçalvesSilvana G P de CamposRejane M GóesWellerson R ScaranoSebastião Roberto TabogaPatricia S L Vilamaior
Published in: The Prostate (2017)
Estrogen was able to slow tumor growth, but the epithelium exhibited features distinct from a normal prostatic epithelium, and this unstable microenvironment could trigger lesion recurrence over time.
Keyphrases
  • stem cells
  • benign prostatic hyperplasia
  • prostate cancer
  • estrogen receptor
  • high glucose
  • diabetic rats
  • replacement therapy
  • drug induced
  • oxidative stress
  • free survival