≥mild PVL after TAVI is common and can be predicted by aortic root calcification volume, larger annulus dimensions, and pre-TAVI transvalvular peak velocity, with calcification volume being an independent predictor for PVL. However, annulus ellipticity, left ventricular outflow tract nontubularity, and diameter-derived prosthesis mismatch had no role in predicting PVL. Importantly, body of leaflet calcifications (versus annulus and tip of leaflet) and cusp calcifications (versus commissure calcification) are more important in predicting PVL. No association between ≥mild PVL and increased risk of all-cause and cardiovascular mortality at 1-year follow-up.
Keyphrases
- aortic valve
- transcatheter aortic valve implantation
- aortic stenosis
- transcatheter aortic valve replacement
- aortic valve replacement
- chronic kidney disease
- left ventricular
- risk factors
- heart failure
- mitral valve
- cardiovascular events
- acute myocardial infarction
- cardiovascular disease
- blood flow
- type diabetes
- coronary artery disease
- pulmonary artery
- percutaneous coronary intervention
- pulmonary hypertension
- atrial fibrillation