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Investigating the Vascular Toxicity Outcomes of the Irreversible Proteasome Inhibitor Carfilzomib.

Panagiotis EfentakisHendrik DoerschmannClaudius WitzlerSvenja SiemerPanagiota-Efstathia NikolaouEfstathios KastritisRoland StauberMeletios Athanasios DimopoulosPhilip WenzelIoanna AndreadouEvangelos Terpos
Published in: International journal of molecular sciences (2020)
Carfilzomib induces a transient vascular impairment and oxidative burst. Cfz+Met increased vascular contractility and synergistically induced autophagy in all settings. Therefore, carfilzomib cannot be accredited for a permanent vascular dysfunction, while Cfz+Met exert vasoprotective potency.
Keyphrases
  • multiple myeloma
  • oxidative stress
  • tyrosine kinase
  • type diabetes
  • endoplasmic reticulum stress
  • diabetic rats
  • high frequency
  • high glucose
  • adipose tissue
  • insulin resistance
  • brain injury
  • weight loss