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LCZ696, an Angiotensin Receptor-Neprilysin Inhibitor, Ameliorates Endothelial Dysfunction in Diabetic C57BL/6 Mice.

Uugantsetseg MunkhjargalDaiju FukudaJuri MaedaTomoya HaraShintaro OkamotoOyunbileg BavuuTakayuki YamamotoMasataka Sata
Published in: Journal of atherosclerosis and thrombosis (2024)
LCZ696 ameliorated diabetes-induced endothelial dysfunction by increasing the bioavailability of ANP. Our findings suggest that LCZ696 has a vascular protective effect in a diabetic model and highlight that it may be more effective than valsartan.
Keyphrases
  • type diabetes
  • wound healing
  • cardiovascular disease
  • high glucose
  • diabetic rats
  • glycemic control
  • high fat diet induced
  • angiotensin ii
  • mouse model
  • insulin resistance
  • binding protein
  • wild type