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Dysregulation of PRMT5 in chronic lymphocytic leukemia promotes progression with high risk of Richter's transformation.

Zachary A HingJanek S WalkerEthan C WhippLindsey BrintonMatthew CannonPu ZhangSteven SherCasey B CempreFiona BrownPorsha L SmithClaudio AgostinelliStefano A PileriJordan N SkinnerKatie WilliamsHannah PhillipsJami ShafferLarry P BeaverAlexander PanKyle ShinCharles T GregoryGulcin H OzerSelen A YilmazBonnie K HarringtonAmy M LehmanLianbo YuVincenzo CoppolaPearlly S YanPeggy ScherleMin WangPhilip PitisChaoyi XuKris VaddiSelina Chen-KiangJennifer A WoyachJames S BlachlyLapo AlinariYiping YangJohn C ByrdRobert A BaiocchiBradley W BlaserRosa Lapalombella
Published in: Nature communications (2023)
Richter's Transformation (RT) is a poorly understood and fatal progression of chronic lymphocytic leukemia (CLL) manifesting histologically as diffuse large B-cell lymphoma. Protein arginine methyltransferase 5 (PRMT5) is implicated in lymphomagenesis, but its role in CLL or RT progression is unknown. We demonstrate herein that tumors uniformly overexpress PRMT5 in patients with progression to RT. Furthermore, mice with B-specific overexpression of hPRMT5 develop a B-lymphoid expansion with increased risk of death, and Eµ-PRMT5/TCL1 double transgenic mice develop a highly aggressive disease with transformation that histologically resembles RT; where large-scale transcriptional profiling identifies oncogenic pathways mediating PRMT5-driven disease progression. Lastly, we report the development of a SAM-competitive PRMT5 inhibitor, PRT382, with exclusive selectivity and optimal in vitro and in vivo activity compared to available PRMT5 inhibitors. Taken together, the discovery that PRMT5 drives oncogenic pathways promoting RT provides a compelling rationale for clinical investigation of PRMT5 inhibitors such as PRT382 in aggressive CLL/RT cases.
Keyphrases
  • chronic lymphocytic leukemia
  • diffuse large b cell lymphoma
  • transcription factor
  • gene expression
  • clinical trial
  • small molecule
  • nitric oxide
  • type diabetes
  • metabolic syndrome
  • amino acid
  • protein protein