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The role of Src & ERK1/2 kinases in inspiratory resistive breathing induced acute lung injury and inflammation.

Dimitrios ToumpanakisVyronia VassilakopoulouIoanna SigalaPanagiotis ZacharatosIoanna VrailaVassiliki KaravanaStamatios TheocharisTheodoros Vassilakopoulos
Published in: Respiratory research (2017)
Src and ERK1/2 are activated in the lung following IRB and participate in IRB-induced lung injury.
Keyphrases
  • high glucose
  • signaling pathway
  • diabetic rats
  • tyrosine kinase
  • cell proliferation
  • pi k akt
  • lipopolysaccharide induced
  • lps induced
  • inflammatory response