The role of Src & ERK1/2 kinases in inspiratory resistive breathing induced acute lung injury and inflammation.
Dimitrios ToumpanakisVyronia VassilakopoulouIoanna SigalaPanagiotis ZacharatosIoanna VrailaVassiliki KaravanaStamatios TheocharisTheodoros VassilakopoulosPublished in: Respiratory research (2017)
Src and ERK1/2 are activated in the lung following IRB and participate in IRB-induced lung injury.