The role of Src & ERK1/2 kinases in inspiratory resistive breathing induced acute lung injury and inflammation.

Dimitrios ToumpanakisVyronia VassilakopoulouIoanna SigalaPanagiotis ZacharatosIoanna VrailaVassiliki KaravanaStamatios TheocharisTheodoros Vassilakopoulos

Published in: Respiratory research (2017)

Src and ERK1/2 are activated in the lung following IRB and participate in IRB-induced lung injury.

Keyphrases

high glucose
signaling pathway
diabetic rats
tyrosine kinase
cell proliferation
pi k akt
lipopolysaccharide induced
lps induced
inflammatory response