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MyD88 overexpression deteriorates Ang-II-induced ED via upregulating MPO and COX2 and downregulating eNOS in the corpus cavernosum of rats.

Haishan ZhangShujuan ZhangLinpei JiaHai Li
Published in: Journal of cellular biochemistry (2018)
MyD88 overexpression deteriorates Ang-II-induced ED via upregulating MPO and COX2 and downregulating eNOS in the corpus cavernosum rats.
Keyphrases
  • high glucose
  • emergency department
  • endothelial cells
  • diabetic rats
  • angiotensin ii
  • toll like receptor
  • nitric oxide synthase
  • drug induced
  • immune response
  • inflammatory response