Login / Signup

Retinoic acid generates a beneficial microenvironment for liver progenitor cell activation in acute liver failure.

Sai WangFrederik LinkStefan MunkerWenjing WangRilu FengRoman LiebeYujia LiYe YaoHui LiuChen ShaoMatthias P A EbertHui-Guo DingSteven DooleyHong-Lei WengShan-Shan Wang
Published in: Hepatology communications (2024)
During ALF, RA secreted by activated HSCs promotes LPC activation, a prerequisite for subsequent LPC-mediated liver regeneration.
Keyphrases
  • liver failure
  • hepatitis b virus
  • stem cells
  • rheumatoid arthritis
  • disease activity
  • intensive care unit
  • ankylosing spondylitis
  • interstitial lung disease
  • systemic sclerosis