Parsing multiomics landscape of activated synovial fibroblasts highlights drug targets linked to genetic risk of rheumatoid arthritis.
Haruka TsuchiyaMineto OtaShuji SumitomoKazuyoshi IshigakiAkari SuzukiToyonori SakataYumi TsuchidaHiroshi InuiJun HiroseYuta KochiYuho KadonoKatsuhiko ShirahigeSakae TanakaKazuhiko YamamotoKeishi FujioPublished in: Annals of the rheumatic diseases (2020)
Our findings established the dynamic landscape of activated SFs and yielded potential therapeutic targets associated with genetic risk of RA.