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Hyperoxia impairs induced pluripotent stem cell-derived endothelial cells and drives an atherosclerosis-like transcriptional phenotype.

Sean M CarrKatherine OwsianyOttis ScrivnerDylan McLaughlinHanjoong JoLuke P BrewsterKatherine E Hekman
Published in: JVS-vascular science (2024)
Taken together, our studies demonstrate that physiological 4% oxygen culture conditions were sufficient to improve mitochondrial function in high passage cells, but alone was insufficient to preserve angiogenic capacity. Furthermore, late passage cells under typical conditions take on an endothelial-to-mesenchymal phenotype with similarities to ECs found in atherosclerosis.
Keyphrases
  • endothelial cells
  • induced apoptosis
  • cell cycle arrest
  • high glucose
  • cardiovascular disease
  • stem cells
  • gene expression
  • cell death
  • transcription factor
  • type diabetes
  • drug induced
  • diabetic rats