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Aquaporin 4 differentially modulates osmotic effects on vasopressin neurons in rat supraoptic nucleus.

Xiaoran WangTong LiYang LiuShuwei JiaXiaoyu LiuYunhao JiangPing WangVladimir ParpuraYu-Feng Wang
Published in: Acta physiologica (Oxford, England) (2021)
These findings indicate that osmotically driven increase in VP neuronal activity requires the activation of AQP4, which determines a retraction of GFAP filaments.
Keyphrases
  • spinal cord
  • oxidative stress
  • cerebral ischemia
  • subarachnoid hemorrhage