Microglial-induced apoptosis is potentially responsible for hyperalgesia variations during CFA-induced inflammation.
Mansoureh BaniasadiHoma ManahejiNader MaghsoudiSamira DanyaliZahra ZakeriAmirabbas MaghsoudiJalal ZaringhalamPublished in: Inflammopharmacology (2019)
These results suggest that microglial-mediated pain following CFA injection might be related in part to increased spinal cell apoptosis which probably is mediated by PTEN/PI3K/Akt deregulation.
Keyphrases
- pi k akt
- induced apoptosis
- signaling pathway
- neuropathic pain
- cell proliferation
- oxidative stress
- spinal cord
- endoplasmic reticulum stress
- cell cycle arrest
- inflammatory response
- lipopolysaccharide induced
- diabetic rats
- lps induced
- chronic pain
- spinal cord injury
- pain management
- high glucose
- ultrasound guided
- drug induced