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MFN2 overexpression in skeletal muscles of young and old mice causes a mild hypertrophy without altering mitochondrial respiration and H 2 O 2 emission.

Marina CefisManon DargegenVincent MarcangeliShima TaherkhaniMaude DulacJean-Philippe Leduc-GaudetDominique MayakiSabah N A HussainGilles Gouspillou
Published in: Acta physiologica (Oxford, England) (2024)
MFN2 overexpression may be a viable approach to mitigate aging-related muscle atrophy and may have applications for other muscle disorders.
Keyphrases
  • skeletal muscle
  • cell proliferation
  • transcription factor
  • oxidative stress
  • high fat diet induced
  • middle aged
  • metabolic syndrome
  • insulin resistance