Complement C3a and C3a Receptor Activation Mediates Podocyte Injuries in the Mechanism of Primary Membranous Nephropathy.

Published in: Journal of the American Society of Nephrology : JASN (2022)

C3a anaphylatoxin is a crucial effector of complement-mediated podocyte damage in MN. The C3aR antagonist may be a potentially viable treatment for this disease.

Keyphrases

diabetic nephropathy
dendritic cells
regulatory t cells
room temperature
combination therapy
smoking cessation