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Prenatal inflammation reprograms hyperactive ILC2s that promote allergic lung inflammation and airway dysfunction.

Diego A LopezAleah GriffinLorena Moreno AguilarCassandra-Deering RiceElizabeth J MyersKristi J WarrenRobert S WelnerAnna E Beaudin
Published in: bioRxiv : the preprint server for biology (2023)
Prenatal inflammation programs asthma susceptibility by inducing the production of hyperactivated ILC2s in the developing lung.
Keyphrases
  • oxidative stress
  • pregnant women
  • lung function