Focal adhesion kinase and Src mediate microvascular hyperpermeability caused by fibrinogen- γC- terminal fragments.
Xiaohua GuoRebecca A EitnierRichard S BeardJamie E MeeganXiaoyuan YangAlexandra M AponteFang WangPeter R NelsonMack H WuPublished in: PloS one (2020)
The FAK-Src pathway contributes to γC-induced microvascular barrier dysfunction, junction protein phosphorylation and disorganization in a manner that involves RhoA and stress-fiber formation.