Hesperetin suppresses LPS/high glucose-induced inflammatory responses via TLR/MyD88/NF-κB signaling pathways in THP-1 cells.
Aeri LeeHyunJi GuMin-Hee GwonJung-Mi YunPublished in: Nutrition research and practice (2021)
Our results suggest that hesperetin may be a potential agent for suppressing inflammation in diabetes.
Keyphrases
- high glucose
- signaling pathway
- induced apoptosis
- endothelial cells
- pi k akt
- toll like receptor
- inflammatory response
- cell cycle arrest
- oxidative stress
- epithelial mesenchymal transition
- type diabetes
- cardiovascular disease
- immune response
- nuclear factor
- glycemic control
- endoplasmic reticulum stress
- anti inflammatory
- risk assessment
- human health
- diabetic rats
- cell death
- weight loss
- adipose tissue
- drug induced
- insulin resistance
- stress induced