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Cytoskeletal and inter-cellular junction remodelling in endometrial organoids under oxygen-glucose deprivation: a new potential pathological mechanism for thin endometria.

TianLiu PengShuo YangWeisi LianXiaojuan LiuPing ZhengXunsi QinBaoying LiaoPing ZhouYue WangFenting LiuZi YangZhenhong YeHongying ShanXiyao LiuYang YuRong Li
Published in: Human reproduction (Oxford, England) (2024)
This study was supported by the National Natural Science Foundation of China (81925013); National Key Research and Development Project of China (2022YFC2702500, 2021YFC2700303, 2021YFC2700601); the Capital Health Research and Development Project (SF2022-1-4092); the National Natural Science Foundation of China (82288102, 81925013, 82225019, 82192873); Special Project on Capital Clinical Diagnosis and Treatment Technology Research and Transformation Application (Z211100002921054); the Frontiers Medical Center, Tianfu Jincheng Laboratory Foundation(TFJC2023010001). The authors declare that no competing interests exist.
Keyphrases
  • quality improvement
  • public health
  • type diabetes
  • metabolic syndrome
  • endometrial cancer
  • climate change
  • human health
  • induced pluripotent stem cells