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Endoplasmic reticulum stress contributes to cisplatin-induced chronic kidney disease via the PERK-PKCδ pathway.

Shaoqun ShuHui WangJiefu ZhuYing FuJuan CaiAnqun ChenChengyuan TangZheng Dong
Published in: Cellular and molecular life sciences : CMLS (2022)
These results indicate that ER stress contributes to chronic kidney pathologies following cisplatin chemotherapy via the PERK-PKCδ pathway.
Keyphrases
  • endoplasmic reticulum stress
  • induced apoptosis
  • chronic kidney disease
  • end stage renal disease
  • locally advanced
  • squamous cell carcinoma
  • radiation therapy
  • signaling pathway
  • endoplasmic reticulum