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HDACi-dependent microenvironmental normalization overcomes tumor burden-induced T-cell exhaustion.

Andrew NguyenDominique BrownRamya KrishnanDonald J BastinLi DengLan ChenOmar SalemScott R WalshJonathan L BramsonYonghong Wan
Published in: Clinical cancer research : an official journal of the American Association for Cancer Research (2023)
Tumor burden-induced pathway changes can be reversed through epigenetic reprogramming, enabling the conversion from T-cell exhaustion to effector lineage differentiation.
Keyphrases
  • high glucose
  • diabetic rats
  • dna methylation
  • drug induced
  • dendritic cells
  • risk factors
  • regulatory t cells
  • oxidative stress
  • endothelial cells
  • single cell
  • cell fate