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Plasma Fucosylated Glycans and C-Reactive Protein as Biomarkers of HNF1A-MODY in Young Adult-Onset Nonautoimmune Diabetes.

Agata JuszczakTamara PavićFrano VučkovićAmanda J BennettNeha ShahEdita Pape MedvidovićChristopher J GrovesMario ŠekerijaKyla ChandlerCarla BurrowsNataša Rojnić PutarekMarijana Vučić LovrenčićJadranka Ćuća KneževićTim J JamesAnna L GloynGordan LaucMark I McCarthyKatharine R OwenOlga Gornik
Published in: Diabetes care (2018)
Half of rare HNF1A sequence variants do not cause MODY. N-glycan profile and hs-CRP could both be used as tools, alone or as adjuncts to existing pathways, for identifying individuals at high risk of carrying a damaging HNF1A allele.
Keyphrases
  • nuclear factor
  • type diabetes
  • cardiovascular disease
  • cell surface
  • toll like receptor
  • copy number
  • middle aged
  • dna methylation
  • skeletal muscle
  • amino acid
  • weight loss