Cathodal tDCS exerts neuroprotective effect in rat brain after acute ischemic stroke.
Ke-Ying ZhangGang RuiJun-Ping ZhangLing GuoGuang-Zhou AnJia-Jin LinWei HeGui-Rong DingPublished in: BMC neuroscience (2020)
Our results suggested that cathodal tDCS could accelerate the recovery of neurologic deficit and brain damage caused by MCAO. The inhibition of neuroinflammation and apoptosis resulted from cathodal tDCS may be involved in the neuroprotective process.
Keyphrases
- transcranial direct current stimulation
- acute ischemic stroke
- cerebral ischemia
- working memory
- oxidative stress
- subarachnoid hemorrhage
- blood brain barrier
- brain injury
- endoplasmic reticulum stress
- traumatic brain injury
- lipopolysaccharide induced
- cell death
- cell cycle arrest
- cognitive impairment
- inflammatory response