The tumor immune microenvironment of esophageal cancer patients with H. pylori infection exhibited an immunosuppressive phenotype. The targeting of Treg cells has potential in immunotherapy for this patient population.
Keyphrases
- oxidative stress
- induced apoptosis
- helicobacter pylori infection
- end stage renal disease
- stem cells
- ejection fraction
- newly diagnosed
- chronic kidney disease
- helicobacter pylori
- peritoneal dialysis
- prognostic factors
- cell cycle arrest
- cancer therapy
- drug delivery
- cell death
- signaling pathway
- climate change
- endoplasmic reticulum stress
- human health