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Olig1 is required for noggin-induced neonatal myelin repair.

Jennifer K SaboVivi HeineJohn C SilbereisLucas SchirmerSteven W LevisonDavid H Rowitch
Published in: Annals of neurology (2017)
These findings indicate that Olig1 has a critical function in regulation of postnatal neural progenitor cell production in response to Noggin. Ann Neurol 2017;81:560-571.
Keyphrases
  • high glucose
  • diabetic rats
  • preterm infants
  • drug induced
  • white matter
  • oxidative stress