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Identification of the occurrence and potential mechanisms of heterotopic ossification associated with 17-beta-estradiol targeting MKX by bioinformatics analysis and cellular experiments.

Yunpeng ZhangJingwei ZhangChenyu SunFan Wu
Published in: PeerJ (2022)
The compounds Pro-Tyr and 17-beta-Estradiol may bind to MKX and thus affect the interaction of MKX with SIN3A/HDAC1.
Keyphrases
  • bioinformatics analysis
  • estrogen receptor
  • risk assessment
  • cancer therapy
  • anti inflammatory
  • histone deacetylase
  • human health
  • climate change