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Deletion of a Single Lysine Residue at Position 292 of CAMK2A Disrupts Protein Function, Causing Severe Epileptic Encephalopathy and Intellectual Disability.

Carla LintasAngelo FacchianoAlessia AzzaràIlaria CassanoClaudio TabolacciCinzia GalassoFiorella Gurrieri
Published in: Genes (2023)
We discuss our variant in relation to previously reported variants and with the objective of delineating possible genotype-phenotype correlations.
Keyphrases
  • intellectual disability
  • early onset
  • amino acid
  • autism spectrum disorder
  • copy number
  • protein protein
  • binding protein
  • drug induced
  • genome wide