Bni5 tethers myosin-II to septins to enhance retrograde actin flow and the robustness of cytokinesis.

Hiroki OkadaXi ChenKangji WangJoseph MarquardtErfei Bi

Published in: bioRxiv : the preprint server for biology (2023)

Okada et al. have determined the molecular mechanism underlying the Bni5 interactions with septins and myosin-II at the cell division site and uncovered its roles in promoting retrograde actin flow and the robustness of cytokinesis in budding yeast.

Keyphrases

binding protein
single cell
cell migration
cell therapy
saccharomyces cerevisiae
stem cells
cell wall