Simultaneous targeting of oxidative stress and fibrosis reverses cardiomyopathy-induced ventricular remodelling and dysfunction.
Chao WangTracey A GaspariDorota FerensIresha SpizzoBarbara K Kemp-HarperChrishan S SamuelPublished in: British journal of pharmacology (2021)
These findings demonstrated that the simultaneous targeting of oxidative stress and fibrosis is key to treating the pathophysiology and dysfunction induced by cardiomyopathy.