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Deficiency of Intestinal α1-2-Fucosylation Exacerbates Ethanol-Induced Liver Disease in Mice.

Rongrong ZhouCristina LlorenteJinling CaoBei GaoYi DuanLu JiangYanhan WangVipin KumarPeter StärkelLars BodeXuegong FanBernd Schnabl
Published in: Alcoholism, clinical and experimental research (2020)
Intestinal α1-2-fucosylation acts as a host-protective mechanism against EtOH-induced liver disease. 2'-FL is an oligosaccharide naturally present in human milk that could be considered as therapeutic agent for alcohol-associated liver disease.
Keyphrases
  • human milk
  • high glucose
  • diabetic rats
  • low birth weight
  • endothelial cells
  • oxidative stress
  • metabolic syndrome
  • skeletal muscle
  • preterm infants
  • high fat diet induced
  • replacement therapy
  • wild type