Maternal Fructose Intake Increases Liver H2 S Synthesis but Exarcebates its Fructose-Induced Decrease in Female Progeny.
Elena FausteSilvia RodrigoRodrigo AguirreCristina DonisLourdes RodríguezJuan J Álvarez-MillánMaría I PanaderoPaola OteroCarlos BocosPublished in: Molecular nutrition & food research (2020)
Maternal fructose intake produces a fetal programming that increases hepatic H2 S production and, in contrast, exacerbates its fructose-induced drop in female progeny.