Defective formyl peptide receptor 2/3 and annexin A1 expressions associated with M2a polarization of blood immune cells in patients with chronic obstructive pulmonary disease.

Yung-Che ChenMeng-Chih LinChih-Hung LeeShih-Feng LiuChin-Chou WangWen-Feng FangTung-Ying ChaoChao-Chien WuYu-Feng WeiHuang-Chih ChangChia-Cheng TsenHung-Chen Chennull null

Published in: Journal of translational medicine (2018)

Our findings provide evidence for defective FPR2/3 and annexin A1 expressions that, associated with decreased M2a polarization, might be involved in the development of cigarette smoking induced persistent airflow limitation in COPD.

Keyphrases

chronic obstructive pulmonary disease
high glucose
diabetic rats
lung function
binding protein
air pollution