HDAC2 hyperexpression alters hippocampal neuronal transcription and microglial activity in neuroinflammation-induced cognitive dysfunction.
Xiao-Yu SunTeng ZhengXiu YangLe LiuShen-Shen GaoHan-Bing XuYu-Tong SongKun TongLi YangYa GaoTong WuJing-Ru HaoChen LuTao MaCan GaoPublished in: Journal of neuroinflammation (2019)
Reversing HDAC2 in hippocampal CaMKII+ neurons exert a neuroprotective effect against neuroinflammation-induced memory deficits.
Keyphrases
- cerebral ischemia
- traumatic brain injury
- lipopolysaccharide induced
- high glucose
- diabetic rats
- lps induced
- subarachnoid hemorrhage
- inflammatory response
- brain injury
- blood brain barrier
- spinal cord
- drug induced
- histone deacetylase
- transcription factor
- oxidative stress
- working memory
- spinal cord injury
- endothelial cells
- temporal lobe epilepsy