Propofol protected against sepsis-induced brain damage, neuroinflammation, neuronal apoptosis and gliosis through the activation of the Nrf2/HO-1 axis to combat ferroptosis.
Keyphrases
- brain injury
- oxidative stress
- cerebral ischemia
- diabetic rats
- cell death
- subarachnoid hemorrhage
- high glucose
- intensive care unit
- acute kidney injury
- septic shock
- drug induced
- traumatic brain injury
- endoplasmic reticulum stress
- cell cycle arrest
- white matter
- resting state
- endothelial cells
- pi k akt
- blood brain barrier
- cognitive impairment
- multiple sclerosis
- inflammatory response
- functional connectivity